BluesBrother
Master
FYI It might be important because type A the second most common type in the US has been found to be the most vulnerable to the chinese flu.
The coronavirus SARS-CoV-2 may latch more easily onto the airway cells of people with type A (42% of US population) blood compared with those with type B (10% of US population) or type O (44% of US population) blood, type AB is (4% of US population) a new study suggests. The findings hint at a possible explanation for why, throughout the pandemic, studies have found those with type A blood are likelier to catch COVID-19 and develop severe symptoms than other blood types.
Laboratory experiments revealed that part of the coronavirus called the "receptor binding domain" (RBD), which directly binds to cells to jump start infection, also grabs onto unique molecules associated with type A blood. These molecules, known as antigens, show up on cells that line the respiratory tract, including the lungs, according to the study, published March 3 in the journal Blood Advances.
In theory, binding to these structures may help the coronavirus enter and infect the airway cells more easily — however, we don't know that for sure yet, the study authors told Live Science.
The coronavirus SARS-CoV-2 may latch more easily onto the airway cells of people with type A (42% of US population) blood compared with those with type B (10% of US population) or type O (44% of US population) blood, type AB is (4% of US population) a new study suggests. The findings hint at a possible explanation for why, throughout the pandemic, studies have found those with type A blood are likelier to catch COVID-19 and develop severe symptoms than other blood types.
Laboratory experiments revealed that part of the coronavirus called the "receptor binding domain" (RBD), which directly binds to cells to jump start infection, also grabs onto unique molecules associated with type A blood. These molecules, known as antigens, show up on cells that line the respiratory tract, including the lungs, according to the study, published March 3 in the journal Blood Advances.
In theory, binding to these structures may help the coronavirus enter and infect the airway cells more easily — however, we don't know that for sure yet, the study authors told Live Science.
